Association of Unc-51-like Kinase 4 (<em>ULK4</em>) with the reactivity of the extended reward system in response to conditioned stimuli

CONCLUSIONS: Our findings provide further evidence in humans that genetic variation in ULK4 may increase the vulnerability to mental disorders, by modulating the extended reward system function. Future studies are needed to confirm the modulation of the extended reward system by ULK4 and to specify the role of this mechanism in the pathogenesis of psychiatric …